https://ogma.newcastle.edu.au/vital/access/ /manager/Index ${session.getAttribute("locale")} 5 https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:34619 Wed 23 Feb 2022 16:04:35 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:10201 Sat 24 Mar 2018 08:11:25 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:46652 p-NMDAR1) and (3) phosphorylated calmodulin-dependent protein kinase II (p-CaMKII). In addition, the content of whole brain malondialdehyde (MDA) as well as activities of superoxide dismutase (SOD) and glutathione peroxidase (GSH-Px) were determined. Treatment with T significantly elevated the number of intact pyramidal cells in hippocampal CA1 region and markedly increased hippocampal protein and mRNA expression levels of p-NMDAR1 and p-CaMK II. Further, T significantly decreased whole brain MDA levels accompanied by elevated activities of SOD and GSH-Px. Data suggest that the protective effects of T on synaptic plasticity in a mouse AD model may be associated with reduction of oxidant stress.]]> Mon 28 Nov 2022 17:29:45 AEDT ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:49513 Fri 19 May 2023 17:23:55 AEST ]]> https://ogma.newcastle.edu.au/vital/access/ /manager/Repository/uon:49487 Fri 19 May 2023 09:34:55 AEST ]]>